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Nucleotide metabolism is key in Parkinson’s disease, study shows

By Sharon Kingman
Staff Writer

Thursday, January 23, 2014
LONDON – A better understanding of the metabolic pathways affected by mitochondrial defects linked to Parkinson’s disease could lead to new ways of preventing further deterioration in people diagnosed with this condition. The findings may even provide new targets for potential anticancer treatments, as some of the same pathways are also essential in rapidly dividing cancer cells.

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