Researchers at New York University Medical Center have discovered that the CCR5 receptor – well known to infectious disease researchers as a co-receptor for HIV entry – is also the receptor that Staphylococcus aureus' leukotoxin ED, or LukED, uses to target specific sets of immune system cells.

Scientists from the National Cancer Institute have found that PARP inhibitors work through other mechanisms in addition to inhibiting the repair of DNA breaks.